Orthodontically Driven Corticotomy: Tissue Engineering to

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9). To neglect this important service would constitute an error of omission and a logical non sequitur. Given that full disclosure of pathosis is de rigueur (and a legal imperative) in any healthcare venue, it can even be considered ethically untenable to overlook periodontal health. Ignoring periodontitis renders lesions vulnerable to exacerbation during accelerated orthodontic therapy because moving teeth in the presence of active periodontitis can pump supragingival pathogens subgingivally, accelerating the permanent loss of supporting periodontal attachment and creating problematic vertical defects.

The wise writer knows that very few universals can be legitimately defended in clinical biology. It seems that the Bell and Levy article merely achieved a consecration of bias and documentation that any overly aggressive surgery is risky. To their credit, however, the authors discussed the thoughtful idea of a ­two-staged procedure reflecting the palatal flap and labial flaps 5 weeks apart. Moreover, it must be acknowledged that Bell and Levy made a significant contribution about how alveolar bone heals, despite their vague and confusing overstatements about what should be done.

The term used for this phenomenon is bone matrix transportation. 1985: Mostafa – Limited objective therapy Mostafa et al. (1985) diagrammed a surgical– orthodontic technique to treat overerupted maxillary molars showing that corticotomy could be used for one or two tooth segments. It was a Köle-like decortication localized to the alveolus of one tooth, an extruded molar. They reported a survey of 15 patients, noting that only the cortex was incised with a surgical bur and osteotome. No explanation was made about the specific nature of the surgery.