By Gad Loebenstein (Editor), John Peter Carr (Editor)
This ebook is a primary try to hyperlink popular plant resistance phenomena with rising suggestions in molecular biology. Resistance phenomena corresponding to the neighborhood lesion reaction, prompted resistance, "green islands" and resistance in a number of crop crops are associated with new details on gene-silencing mechanisms, gene silencing suppressors, flow proteins and plasmodesmatal gating, downstream signalling elements, and so on.
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It could be that alterations in TMV coat protein structure reveal some interaction with a previously unknown R gene that is common to many different plants. However, Bendahmane et al. (1999) suggest that these alterations in coat protein structure may be destabilizing cellular membranes, and this would trigger host cell death. The P6 protein of Cauliflower mosaic virus The P6 protein of CaMV is recognized by resistance genes in D. stramonium, N. bigelovii, N. glutinosa, N. edwardsonii, and Arabidopsis thaliana ecotype Tsu-0 (Agama et al.
And Lovrekovich, L. 1966. Interference with tobacco mosaic virus local lesion formation in tobacco by injecting heat-killed cells of Pseudomonas syringae. Virology 30:587-591. W. 1969. Inhibition of tobacco mosaic virus by a bacterial extract. Phytopathology 59:658-662. , Metraux, J. , and Defago, G. 1994. Induction of systemic resistance of tobacco to tobacco necrosis virus by the root-colonizing Pseudomonas fluorescens strain CHA0: Influence of the gacA gene and of pyoverdine production. Phytopathology 84:139-146.
1997). Two experimental approaches have been used to show that the helicase domain of the replicase is the Avr determinant. Padgett and coworkers (1997) exchanged domains between infectious clones of virulent and avirulent TMV isolates, whereas other groups expressed TMV genes individually by agroinfiltration to identify the helicase domain of the replicase as the HR determinant (Abbink et al. 1998; Erickson et al. 1999). Helicases participate in the replication of the viral nucleic acid by promoting the unwinding of duplex nucleic acids (Hull, 2002; Kadarei and Haenni, 1997).