By J Timothy Wright
This booklet presents the practitioner with a framework for developing a analysis and constructing an appropriate therapy plan in sufferers providing with a variety of developmental defects of tooth. The stipulations coated contain failure of teeth eruption, hypodontia, untimely teeth exfoliation, defects of teeth improvement, and defects of dentin improvement, with complete attention of either syndromic and non-syndromic defects. In every one case the phenotype and genotype are first defined, by means of diagnostic info, together with the supply of genetic trying out, and therapies. Summarizing tables are used to focus on the main diagnostic gains, and necessary illustrated case shows are integrated. Cleft palate is additionally addressed, with info on etiology, phenotypes, remedy timing and ways, and dental administration. The last bankruptcy offers stimulating reflections on power destiny instructions within the prognosis and therapy of those disorders.
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Extra resources for Craniofacial and Dental Developmental Defects: Diagnosis and Management
Sample text
Chlorhexidine is also sometimes used [15, 34]. Langerhans cell histiocytosis is a systemic condition that may first be recognized and diagnosed by the dentist. In some cases the oral changes may be the first pathological sign which may lead the patient to seek a dental evaluation [36]. It has been reported in fact that up to 20 % of the cases have their initial tissue infiltration in the oral cavity, usually the mandible [15]. 3 Conditions Associated with Premature Exfoliation of Primary Teeth 41 Fig.
The primary dentition may also demonstrate delayed eruption and immature root formation. This, along with acute root resorption and alveolar bone loss, results in the mobility and early loss of the primary dentition [1, 7]. Management of the dentition is palliative. Coffin-Lowry Syndrome Children with Coffin-Lowry syndrome (OMIM #303600), an X-linked inherited condition caused by mutations in a growth factor-related gene [1, 8], present with intellectual disabilities and skeletal anomalies [1, 8–10].
This tooth loss is usually spontaneous with the exfoliation being associated with a loss of alveolar bone along with defective or hypoplastic cementum (Fig. 4) [1, 3]. Alkaline phosphatase is critical for normal development of mineralized tissues, and hypophosphatasia results in abnormal cementoblast function and the cell’s ability to regulate pyrophosphates critical for normal tissue formation and mineralization during cementogenesis. This defective cementum results in a weakened tooth to alveolar bone attachment which in turn leads to the loss of the primary dentition [15].