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Eicosanoids and related compounds in plants and animals by A. F. Rowley, Hartmut Kühn, T. Schewe

24 February 2017 adminPlants

By A. F. Rowley, Hartmut Kühn, T. Schewe

Eicosanoids are a various workforce of biologically energetic molecules derived from polyunsaturated fatty acid precursors. This quantity attracts jointly for the 1st time a chain of overviews at the biosynthesis and practical value of those and similar compounds in quite a lot of animals, crops, and micro-organisms. All chapters are written via famous specialists of their fields, and plenty of utilize a great deal of unpublished fabrics. This quantity is geared toward complicated undergraduates and at researchers attracted to lipid biochemistry and normal plant and animal biology.

Originally released in 1999.

The Princeton Legacy Library makes use of the newest print-on-demand expertise to back make on hand formerly out-of-print books from the prestigious backlist of Princeton college Press. those paperback variants look after the unique texts of those vital books whereas offering them in sturdy paperback variations. The aim of the Princeton Legacy Library is to significantly raise entry to the wealthy scholarly historical past present in the hundreds of thousands of books released through Princeton college Press given that its founding in 1905.

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Additional resources for Eicosanoids and related compounds in plants and animals

Example text

Therefore, selective inhibition of C O X -2 activity w ould provide a significant im provem ent over therapeutics fo r inflam m atory diseases. Several arom atic com pounds w ith a m ethyl sulph o n y l group, such as N S-398, N imesulide, T-614, FK3311, Floslide, L-745337, D up-697 and SC-58125 have been developed as C O X -2-specific inhibitors [16]. R ecent studies have identified N S A ID s th a t preferentially inhibit C O X -I o r C O X -2 , o r in h ib it b o th isoform s equally [17-19], T he different affinities tow ard som e fatty acid substrates and N S A ID s suggested a subtle difference betw een the active sites of C O X -I and C O X -2.

T he observation suggests th a t C O X -I and C O X -2 in the E R and C O X -2 in the N E constitute independent prostanoid biosynthetic systems. M ost of the current know ledge on the biological contributions of the tw o C O X isoform s has come from studies by N SA ID inhibition, C O X -2 induction or both. Both gene d isruption and overexpression of the C O X isoform s have recently been developed as alternative approaches to investigate the problem . It has been dem onstrated th at C O X -I gene d isru p tio n reduces arachidonic acid-induced inflam m ation and indom ethacin-induced gastric ulceration in mice [25], O n the o th er hand, renal abnorm alities and an altered inflam m atory response w ere observed in mice w ith C O X -2 gene disruption [26,27], Rat intestinal epithelial cells p erm anently infected w ith a C O X -2 expression vector show ed n o t o nly the elevated expression level of C O X -2 p rotein, b u t also b o th increased adhesion to extracellular matrix and inhibition of apoptosis [28].

Pace-Asciak1J. (1984) J. Biol. Chem. P. M . (1987) Biochem . B iophys. Res. C om m un. , D em in, P. and N igam , S. (1998) in L ipoxygenases and lipoxygenase m etabolites. Biological A ctions (N igam , S. J. R . (1990) J. Biol. Chem. , Wu, P. L. (1990) Proc. N atl. Acad. Sci. A. 87,3037-3041 Spiteller, G. (1993) J. R. P. (1990) J. Biol. Chem. R. H . (1990) FEBS L ett. R. H . (1991) J. Biol. Chem. , M anna, S. R. (1986) Biochem. B iophys. Res. C om m un. L. L. (1983) J. Biol. Chem. F. H . (1986) J.

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